1 |
Sun N, Youle RJ, Finkel T. The Mitochondrial Basis of Aging[J]. Mol Cell, 2016, 61(5): 654-666.
|
2 |
Ahlqvist KJ, Hamalainen RH, Yatsuga S, et al. Somatic progenitor cell vulnerability to mitochondrial DNA mutagenesis underlies progeroid phenotypes in Polg mutator mice[J]. Cell Metab, 2012, 15(1): 100-109.
|
3 |
Larsen SB, Hanss Z, Kruger R. The genetic architecture of mitochondrial dysfunction in Parkinson′s disease[J]. Cell Tissue Res, 2018, 373(1): 21-37.
|
4 |
Ito S, Araya J, Kurita Y, et al. PARK2-mediated mitophagy is involved in regulation of HBEC senescence in COPD pathogenesis[J]. Autophagy, 2015, 11(3): 547-559.
|
5 |
Araya J, Tsubouchi K, Sato N, et al. PRKN-regulated mitophagy and cellular senescence during COPD pathogenesis[J]. Autophagy, 2019, 15(3): 510-526.
|
6 |
Ge Y, Shi X, Boopathy S, et al. Two forms of Opa1 cooperate to complete fusion of the mitochondrial inner-membrane[J]. eLife Sciences, 2020, 9. DOI: 10.7554/eLife.50973
|
7 |
Bernardini JP, Lazarou M, Dewson G. Parkin and mitophagy in cancer[J]. Oncogene, 2017, 36(10): 1315-1327.
|
8 |
Vega RB, Horton JL, Kelly DP. Maintaining ancient organelles: mitochondrial biogenesis and maturation[J]. Circ Res, 2015, 116(11): 1820-1834.
|
9 |
任成山,王关嵩,钱桂生. 慢性阻塞性肺疾病的成因及其治疗的困惑与希望[J/CD]. 中华肺部疾病杂志(电子版), 2019, 12(2): 127-141.
|
10 |
Fang L, Gao P, Bao H, et al. Chronic obstructive pulmonary disease in China: a nationwide prevalence study[J]. Lancet Respir Med, 2018, 6(6): 421-430.
|
11 |
Araya J, Tsubouchi K, Sato N, et al. PRKN-regulated mitophagy and cellular senescence during COPD pathogenesis[J]. Autophagy, 2019, 15(3): 510-526.
|
12 |
Bulua AC, Simon A, Maddipati R, et al. Mitochondrial reactive oxygen species promote production of proinflammatory cytokines and are elevated in TNFR1-associated periodic syndrome (TRAPS)[J]. J Exp Med, 2011, 208(3): 519-533.
|
13 |
Minutoli L, Puzzolo D, Rinaldi M, et al. ROS-Mediated NLRP3 inflammasome activation in brain, heart, kidney, and testis ischemia/reperfusion Injury[J]. Oxid Med Cell Longev, 2016, 2016: 2183026.
|
14 |
Hoffmann RF, Zarrintan S, Brandenbuge SM, et al. Prolonged cigarette smoke exposure alters mitochondrial structure and function in airway epithelial cells[J]. Respir Res, 2013, 14: 97.
|
15 |
Hara H, Araya J, Ito S, et al. Mitochondrial fragmentation in cigarette smoke-induced bronchial epithelial cell senescence[J]. Am J Physiol Lung Cell Mol Physiol, 2013, 305(10): L737-L746.
|
16 |
Jiang Y, Wang X, Hu D. Mitochondrial alterations during oxidative stress in chronic obstructive pulmonary disease[J]. Int J Chron Obstruct Pulmon Dis, 2017, 12: 1153-1162.
|
17 |
Ballwge K, Mutze K, Königshoff M, et al. Cigarette smoke extract affects mitochondrial function in alveolar epithelial cells[J]. Am J Physiol Lung Cell Mol Physiol, 2014, 307(11): L895-L907.
|
18 |
Ahmad T, Sundar IK, Lerner CA, et al. Impaired mitophagy leads to cigarette smoke stress-induced cellular senescence: implications for chronic obstructive pulmonary disease[J]. FASEB J, 2015, 29(7): 2912-2929.
|
19 |
Van der Toorn M, Rezayat D, Kauffman HF, et al. Lipid-soluble components in cigarette smoke induce mitochondrial production of reactive oxygen species in lung epithelial cells[J]. Am J Physiol Lung Cell Mol Physiol, 2009, 297(1): L109-L114.
|
20 |
Fujii S, Hara H, Araya J, et al. Insufficient autophagy promotes bronchial epithelial cell senescence in chronic obstructive pulmonary disease[J]. Oncoimmunology, 2012, 1(5): 630-641.
|
21 |
Ito S, Araya J, Kurita Y, et al. PARK2-mediated mitophagy is involved in regulation of HBEC senescence in COPD pathogenesis[J]. Autophagy, 2015, 11(3): 547-559.
|
22 |
Mizumura K, Cloonan SM, Nakahira K, et al. Mitophagy-dependent necroptosis contributes to the pathogenesis of COPD[J]. J Clin Invest, 2014, 124(9): 3987-4003.
|
23 |
Zhang Z, Cheng X, Yue L, et al. Molecular pathogenesis in chronic obstructive pulmonary disease and therapeutic potential by targeting AMP-activated protein kinase[J]. J Cell Physiol, 2018, 233(3): 1999-2006.
|
24 |
Tang GJ, Wang HY, Wang JY, et al. Novel role of AMP-activated protein kinase signaling in cigarette smoke induction of IL-8 in human lung epithelial cells and lung inflammation in mice[J]. Free Radic Biol Med, 2011, 50(11): 1492-1502.
|
25 |
Wang W, Yang X, López DSI, et al. Increased AMP:ATP ratio and AMP-activated protein kinase activity during cellular senescence linked to reduced HuR function[J]. J Biol Chem, 2003, 278(29): 27016-27023.
|
26 |
Correia-Melo C, Marques FD, Anderson R, et al. Mitochondria are required for pro-ageing features of the senescent phenotype[J]. EMBO J, 2016, 35(7): 724-742.
|
27 |
Vanella L, Li VG, Distefano A, et al. A new antioxidant formulation reduces the apoptotic and damaging effect of cigarette smoke extract on human bronchial epithelial cells[J]. Eur Rev Med Pharmacol Sci, 2017, 21(23): 5478-5484.
|
28 |
Yu G, Tzouvelekis A, Wang R, et al. Thyroid hormone inhibits lung fibrosis in mice by improving epithelial mitochondrial function[J]. Nature Medicine, 2018, 24(1): 39-49.
|